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Compiled by Mazen A. Maktabi, M.D.
University of Iowa, Iowa
City, Iowa
1. Aoki
H, Inoue M, Mizobe T, Harada M, Imai H, Kobayashi A: Platelet function
is inhibited by nitric oxide liberation during nitroglycerin-induced
hypotension anaesthesia. Br J Anaesth. 79: 476-81, 1997.
2. Boldt
J, Weber A, Mailer K, Papsdorf M, Schuster P: Acute normovolaemic
haemodilution vs controlled hypotension for reducing the use of
allogeneic blood in patients undergoing radical prostatectomy. Br
J Anaesth 82: 170-4, 1999.
3. Brodsky
JW, Dickson JH, Erwin WD, Rossi CD: Hypotensive anesthesia for scoliosis
surgery in Jehovahs Witnesses. Spine 16:304-306, 1991.
4. Chang
HS, Hongo K, Nakagawa H: Adverse effects of limited hypotensive
anesthesia on the outcome of patients with subarachnoid hemorrhage.
J Neurosurg. 92: 971-5, 2000.
5. Dietrich
GV, Heesen M, Boldt J, Hempelmann G: Platelet function and adrenoceptors
during and after induced hypotension using nitroprusside. Anesthesiology
85:1334-1340, 1996.
6. Endoh
H, Honda T, Ohashi S, Hida S, Shibue C, Komura N: The influence
of nicardipine-, nitroglycerin-, and prostaglandin E(1)-induced
hypotension on cerebral pressure autoregulation in adult patients
during propofol-fentanyl anesthesia. Anesth Analg 94(1):169-73,
2002.
7. Enlund
M, Andersson J, Hartvig P, Valtysson J, Wiklund L: Cerebral normoxia
in the rhesus monkey during isoflurane- or propofol-induced hypotension
and hypocapnia, despite disparate blood-flow patterns. A positron
emission tomography study. Acta Anaesthesiol Scand 41: 1002-10,
1997.
8. Fukusaki
M, Maekawa T, Kobayashi I, Hara T, Sumikawa K: Catecholamine and
renin-angiotensin response during controlled hypotension induced
by prostaglandin E1 combined with hemodilution during isoflurane
anesthesia. J Clin Anesth 9: 321-7, 1997.
9. Harris
SN, Rinder CS, Rinder HM, Tracey JB, Smith BR, Hines R: Nitroprusside
inhibition of platelet function is transient and reversible by catecholamine
priming. Anesthesiology 83:1145-1152, 1995.
10. Hersey
SL, O'Dell NE, Lowe S, Rasmussen G, Tobias JD, Deshpande JK, Mencio
G, Green N: Nicardipine versus nitroprusside for controlled hypotension
during spinal surgery in adolescents. Anesth Analg 84:1239-1244,
1997.
11. Irestedt
L, Lagerkranser M, Bergstrand G, Gordon E: Cerebral blood flow and
metabolism during adenosine-induced hypotension in patients undergoing
cerebral aneurysm surgery. Acta Anaesthesiol Scand 33:15-28, 1989.
12. Jones
AG: Profound hypotension: ethical considerations. Hosp Med 63(2):92-4,
2002.
13. Kadoi
Y, Saito S, Kunimoto F, Morita T, Goto F, Kawahara F, Fujita N:
Cerebral oxygenation during prostaglandin E1 induced hypotension.
Can J Anaesth 45: 860-4, 1998.
14. Khambatta
HJ, Stone JG, Khan E: Hypertension on discontinuation of sodium
nitroprusside. Anesthesiology 51:127-130, 1979.
15. Khambatta
HJ, Stone JG, Khan E: Propranolol alters renin release during nitroprusside-induced
hypotension and prevents hypertension on discontinuation of nitropruside.
Anesth Analg 60:569-573, 1981.
16. Knight
PR, Lane GA, Hensiger RN, Bolles RS, Bjoraker DG: Catecholamine
and renin-angiotensin response during hypotensive anesthesia induced
by sodium nitroprusside or trimethaphan camsylate. Anesthesiology
59:248-253, 1983.
17. Lam
AM, Gelb AW: Cardiovascular effects of isoflurane induced hypotension
for cerebral aneurysm surgery. Anesth Analg 62:742-748, 1983.
*18. Michenfelder
JD, Milde JH: The interaction of sodium nitroprusside, hypotension,
and isoflurane in determining cerebral vasculature effects. Anesthesiology
69:870-875, 1988.
19. Ohata
K, El-Naggar A, Takami T, Morino M, El-Adawy Y, El-Sheik K, Inoue
Y, Hakuba A: Efficacy of induced hypotension in the surgical treatment
of large cavernous sinus cavernomas. J Neurosurg 90: 702-8, 1999.
20. Owall
A, Lagerkranser M, Sollevi A: Effects of adenosine-induced hypotension
on myocardial hemodynamics and metabolism during cerebral aneurysm
surgery. Anesth Analg 67:228-232, 1988.
21. Shapira
Y, Gurman G, Artru AA, Ousyscher IE, Lam AM, Kollender Y, Meller
I: Combined hemodilution and hypotension monitored with jugular
bulb oxygen saturation, EEG, and ECG decreases transfusion volume
and length of ICU stay for major orthopedic surgery. J Clin Anesth
9: 643-9, 1997.
22. Sperry
RJ, Monk CR, Durieux ME, Longnecker DE: The influence of hemorrhage
on organ perfusion during deliberate hypotension in rats. Anesthesiology
77:1171-1177, 1992.
23. Suttner
SW, Lang K, Boldt J, Kumle B, Maleck WH, Piper SN: The influence
of hyperoxic ventilation during sodium nitroprusside-induced hypotension
on skeletal muscle tissue oxygen tension. Anesthesiology 96(5):1103-8,
2002.
24. Suttner
SW, Piper SN, Lang K, Huttner I, Kumle B, Boldt J: Cerebral effects
and blood sparing efficiency of sodium nitroprusside-induced hypotension
alone and in combination with acute normovolaemic haemodilution.
Br J Anaesth 87(5):699-705, 2001.
+25. Tinker
JH, Michenfelder JD: Sodium nitroprusside: pharmacology, toxicology,
and therapeutics. Anesthesiology 45:340-354, 1976.
26. Turner
JM, Powell D, Gibson RM, McDowall DG: Intracranial pressure changes
in neurosurgical patients during hypotension induced with sodium
nitroprusside or trimetaphan. Br J Anaesth 49:419-425, 1977.
+27. Van Aken H, Miller
ED Jr: Deliberate hypotension, Anesthesia. Edited by Miller
RD. New York,
Churchill Livingstone, 1481-1503, 1994.
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